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Kaur, Simranjit
- Pesticide Toxicity and Avifauna of Punjab
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Authors
Affiliations
1 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
2 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
1 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
2 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
Source
The Asian Journal of Animal Science, Vol 10, No 1 (2015), Pagination: 74-80Abstract
Use of pesticides, in modern agriculture has increased tremendously. Pesticides affect the human, environment and wildlife including birds. Three main groups of chemical synthetic pesticides are organochlorine, organophosphate and carbamate. Because of persistent nature, organochlorine are no longer in use in several countries. But some of them like aldrin, dieldrin, lindane and endosulfan are still in use in developing countries. They cause widespread population decline of raptorial birds like the peregrine falcon, the sparrow hawk and bald eagle. The well known effect of DDT in eggshell thinning of the peregrine falcon is caused by its highly persistent metabolite DDE [1,1, bis-4-chlorphenyl]-2,2 dichlorethylene]. Organophosphate and carbamate insecticides do not bioaccumulate in the food chains and are less persistent. They have replaced the more persistent organochlorines. This communication elaborates the effect of synthetic chemical pesticides on birds along with a note on policy framework on use of pesticides.Keywords
Birds, Organochlorines, Acute and Sub Lethal Effects.
- Excretory Pattern and Characteristics of Excreta of Blue Rock Pigeon (Columba livia)
Abstract Views :214 |
PDF Views:0
Authors
Affiliations
1 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
2 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
1 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
2 Department of Zoology, Punjab Agricultural University, LUDHIANA (PUNJAB), IN
Source
The Asian Journal of Animal Science, Vol 11, No 1 (2016), Pagination: 78-80Abstract
No Abstract.Keywords
Excretory Pattern, Excreta, Blue Rock Pigeon.References
- Dauwe, T., Janssens, E., Pinxten, R. and Eens, M. (2005). The reproductive success and quality of blue tits (Parus caeruleus) in a heavy metal pollution gradient. Environ. Pollut., 136 : 243-251.
- DeCant, J. and Barrett, M. (2010). Environmental fate and ecological risk assessment for the registration of clothianidin for use as a seed treatment on mustard seed (oilseed and condiment) and cotton. United States Environmental Protection Agency, WASHINGTON, D.C.
- Goodwin, D. (1983). Pigeons and doves of the world. Ithaca: Comstock Publishing Associates.
- Detection of Sink Hole Attacks in MANET
Abstract Views :117 |
PDF Views:4
Authors
Source
International Journal of Innovative Research and Development, Vol 3, No 2 (2014), Pagination:Abstract
A mobile ad-hoc network (MANET) is most popular example of wireless mobile communication. This network set up by the collection of multi-hop and self-configured mobile nodes without any network infrastructure. Topology of these network changes rapidly and unpredictably. Since mobile nodes are communicating with each other and willing to forward packets to other nodes in the network. These types of networks are more vulnerable to various security threats. One of the major security attacks in MANET is denial of service (DoS) attack. This paper concentrates on the study of the effects of two types of DoS attacks, namely Gray hole attack and Black hole attack in MANET using the reactive routing protocol and also provide the solution to black hole attack. All experiments are simulated for Gray Hole attacks, for black hole attack and for a solution under a variety of ad hoc network conditions such as throughput, packet deliveryratio and normalized routing load inorder to understand the severity of this attack. All simulation is done by network simulator (NS-2).
Keywords
MANET, AODV, gray hole attack, black hole attack, IDSFull Text
- Neonatal Diabetes – A Case Report
Abstract Views :217 |
PDF Views:1
Authors
Affiliations
1 Dept. of Paediatrics, GMCH 32, Chandigarh, IN
2 Assam Medical College, Dibrugarh, IN
1 Dept. of Paediatrics, GMCH 32, Chandigarh, IN
2 Assam Medical College, Dibrugarh, IN
Source
The Indian Practitioner, Vol 71, No 8 (2018), Pagination: 32-34Abstract
Neonatal diabetes mellitus (NDM), defined as persistent hyperglycaemia occurring in the first six months of life, is a rare cause of hyperglycaemia. We report a case of a four-month-old girl child with diabetes presented in Diabetic ketoacidosis (DKA) with severe dehydration, respiratory distress and encephalopathy. The child gradually improved with insulin and fluid therapy. There have been various studies showing the incidence of neonatal diabetes to be around 0.5%. This rare incidence of the disease makes it important to consider neonatal diabetes as an important differential in case of any child presenting with severe dehydration, acidosis and encephalopathy, especially in age group less than 6 months, where the chances of getting it missed are high.Keywords
Neonatal Diabetes, DKA, Sulfonylureas, Subcutaneous Insulin, ABCC8, KCNJ11 Mutations, Transient Diabetes, Permanent Diabetes.References
- White, P.: Diabetes in Childhood and Adolescence, Philadelphia, 1932, Lea and Febiger. 230 The Journal of Pediatrics.
- Joslin, E. P., Root, –I. F., White, P., and Marble, A.: The Treatment of Diabetes Mellitus, Philadelphia, 1952, Lea and lebiger, pp. 666–668.
- John, Henry J.: Diabetes Mellitus in Children, J. PEDIAT. 35: 723, 1949.
- Engelson, Gunnar, and Zettcrqvist, Per: Congenital Diabetes Mellitus and Neonatal Pseudodiabetes Mellitus, Arch. Dis. Child. 32: 193, 1957.
- De Franco E, Flanagan SE, Houghton JAL, Lango Allen H, Mackay DJG, Temple IK, et al. The effect of early, comprehensive genomic testing on clinical care in neonatal diabetes: an international cohort study. Lancet (London, England) 2015;386:957–963. doi: 10.1016/S0140–6736(15)60098– [PMC free article][PubMed] [Cross Ref]
- Iafusco D, Massa O, Pasquino B, Colombo C, Iughetti L, Bizzarri C, et al. Minimal incidence of neonatal/infancy onset diabetes in Italy is 1:90,000 live births. Acta Diabetol. 2012;49:405–408. doi: 10.1007/s00592–011–0331–. [PMC free article] [PubMed] [Cross Ref]
- Slingerland AS, Shields BM, Flanagan SE, Bruining GJ, Noordam K, Gach A, et al. Referral rates for diagnostic testing support an incidence of permanent neonatal diabetes in three European countries of at least 1 in 260,000 live births. Diabetologia. 2009;52:1683–1685. doi: 10.1007/s00125–009–1416–6. [PMC free article] [PubMed] [Cross Ref]
- Ahn SY, Kim G–H, Yoo H–W. Successful sulfonylurea treatment in a patient with permanent neonatal diabetes mellitus with a novel KCNJ11 mutation. Korean J. Pediatr. 2015;58:309–312. doi:10.3345/kjp.2015.58.8.309. [PMC free article] [PubMed] [Cross Ref]
- Proks P, Arnold AL, Bruining J, Girard C, Flanagan SE, Larkin B, et al. A heterozygous activating mutation in the sulphonylurea receptor SUR1 (ABCC8) causes neonatal diabetes. Hum Mol Genet. 2006;15:1793–1800. doi: 10.1093/hmg/ddl101. [PubMed] [Cross Ref]
- Gloyn AL, Pearson ER, Antcliff JF, Proks P, Bruining GJ, Slingerland AS, et al. Activating mutations in the gene encoding the ATP–sensitive potassium–channel subunit Kir6.2 and permanent neonatal diabetes. N Engl J Med. 2004;350:1838–1849. doi: 10.1056/NEJMoa032922. [PubMed] [Cross Ref]
- Babenko AP, Polak M, Cavé H, Busiah K, Czernichow P, Scharfmann R, et al. Activating mutations in the ABCC8 gene in neonatal diabetes mellitus. N Engl J Med. 2006;355:456–466. doi:10.1056/NEJMoa055068. [PubMed] [Cross Ref]
- Rubio–Cabezas O, Ellard S. Diabetes mellitus in neonates and infants: genetic heterogeneity, clinical approach to diagnosis, and therapeutic options. Horm Res pædiatrics. 2013;80:137–146. doi: 10.1159/000354219. [PMC free article] [PubMed] [Cross Ref]
- Vaxillaire M, Dechaume A, Busiah K, Cavé H, Pereira S, Scharfmann R, et al. New ABCC8 mutations in relapsing neonatal diabetes and clinical features. Diabetes. 2007;56:1737–1741. doi: 10.2337/db06–1540. [PubMed] [Cross Ref]
- Rubio–Cabezas O, Hattersley AT, Njølstad PR, Mlynarski W, Ellard S, White N, et al. ISPAD Clinical Practice Consensus Guidelines 2014. The diagnosis and management of monogenic diabetes in children and adolescents. Pediatr. Diabetes. 2014; 15(Suppl 2):47–64. [PubMed]
- Wright NM, Metzer DL, Borowltz SM, Clarke WL. Permanent neonatal diabetes mellitus and pancreatic insufficiency resulting from congenital pancreatic agen–esis. Am J Dis Child 1993: 147: 607–690.
- Aynsley–Green A, Hawdon JM. Metabolic Disease. In: Textbook of Neonatology, Rennie JM, Roberton NRC (eds) 3rd edn. Edinburg: Churchill Livingstone 1999, pp. 939–956.
- Graves’ Disease In Patient With Down’s Syndrome – Not So Uncommon
Abstract Views :155 |
PDF Views:0
Authors
Affiliations
1 Dept. of Paediatrics, GMCH 32, Chandigarh, IN
2 AMC, Dibrugarh, IN
1 Dept. of Paediatrics, GMCH 32, Chandigarh, IN
2 AMC, Dibrugarh, IN
Source
The Indian Practitioner, Vol 71, No 9 (2018), Pagination: 46-48Abstract
Down’s syndrome (DS) is one of the most common chromosomal disorders (one in 800 births) and the most common cause of mental retardation. It is well known that patients with Down’s syndrome have an increased prevalence of autoimmune disorders, affecting both endocrine and non-endocrine organs. The most common autoimmune disease in Down’s syndrome is related to the thyroid gland. Hyperthyroidism is also found at a higher-than-average rate in patients with DS, although only isolated cases have been reported. We here report a case of a 10-year-old female child showing a rare association between Graves’ and Down’s syndrome. The child had symptoms of anxiety, weight loss and swelling in neck. The symptoms gradually improved with methamizole.Keywords
Down’s Syndrome, Graves’ Diseases, Thyroid, Autoimmune Disease.References
- Hassold Tj, Jacobs PA (1984) Trisomy in man, Annu Rev Genet 18: 69-97
- Rohrer TR, Hennes P, Thon A, Dost A, Grabert M, et al. (2010). Down’s syndrome in diabetic patients ages < 20 years: an analysis of metabolic status, glycaemic control and autoimmunity in comparison with type 1 diabetes. Diabetologia 53:1070-1075.
- Fort P, Lifshitz F, Bellisario R, et al. Abnormalities of thyroid function in infants with Down syndrome. J Pediatr 1984;104:545–9.
- Pueschel SM, Pezzullo JC. Thyroid dysfunction in Down syndrome. Am J Dis Child 1985;139:636–9
- Gershwin ME, Crinella FM, Castles JJ, Trent JKT. Immune characteristics of Down’s syndrome. J MentDefic Res 1977;21 :237-49.
- De Luca F, corrias A, Salderno M, Wasniewska M, Gastaldi R, et al. (2010) Peculiarities of Graves’ disease in children and adolescent with Down’s syndrome. Eur J Endocrinol 162:591-595
- Wang S, Mao S, Zhao G, Wu H (2000) relationship between estrogen receptor and Graves’ disease, ZhonghuaWaiKeZaZhi 38:619-621
- Kisiel B, Bednarczuk t, Kostrzewa G, KosiA, ska J, MiAkiewicz P, et al. (2008) Polymorphism of the oestrogen receptor beta gene (ESR2) is associated with susceptibility of Graves’ disease. Clin Endocrinal (Oxf) 68: 429-434
- Goday- Arno A, Cerda- Esteva m, Flores – Le- Roux JA, Chillaron – Jordan JJ, Corretger JM, et al. (2009) Hyperthyroidism in a population with Down Syndrome (DS). ClinEndocrinol (Oxf) 71:110-114
- American Academy of Pediatrics, Committee on Genetics. Health supervision for children with Down syndrome. Pediatrics. 2001;107:442–9
- Kaguelidou F, Alberti C, Castanet M, Guitteny MA, Czernichow P, Leger J for the French Childhood Graves’ Disease Study Group. Predictors of autoimmune hyperthyroidism relapse in children after discontinuation of antithyroid drug treatment. J ClinEndocrinolMetab 2008; 93: 3817-26.